Meeting suddenly turn hostile chronic second liver

Meeting suddenly turn hostile chronic second liver change face be our country Sichuan opera is absolutely vivid, actor stagewise one face about changes give a pair of new types of facial makeup in operas, liver of how chronic second is also met suddenly turn hostile ? Does it change how? Suddenly turn hostile one: Hide a gender He Xiang of chronic second liver is long this year 42 years old, energetic, because work in rural basic level, compare all the time busy, not quite notice the body. Because appetite is unwell recently, without interest, do not love to have a meal, must go to a hospital to check, this ability discovers, his hepatic function had a change. Especially transaminase (ALT) is apparently elevatory. Exsanguinate assay looks, not be small 3 this world, also not be big 3 this world, fight one HBs however, fight one HBe and fight one HBc positive. This is the 3 big antibody of second liver, especially the first antibody (fighting one HBs) is protection antibody, that is to say, in the blood of a person the examination gives this kind of antibody, often hint this person produced immune power to second liver. So, whether be other model hepatitis? The mark matter that the doctor investigated virus of armour, third, fourth, the fifth of the ten Heavenly Stem hepatitis repeatedly, the result is electronegative. Say, is He Xiang's long hepatitis current foregone armour, second, third, fourth ' , the fifth of the ten Heavenly Stem hepatitis? He Xiang grows not to take other remedy basically at ordinary times, not be sexual liver damages medicaments for certain, the hepatitis with scarce what be? Be afraid of bungle illness, improve only courtyard. Come to city class hospital, the doctor grows to He Xiang checked nucleic acid of virus of serum second liver. Namely HBVDNA. The result after two days comes out, it is electropositive, the answer was found, he Xiang is long is hide a gender chronic second liver (the name) that medical experts gave it to remove an afford for thought. Common sense tells us, in the blood of patient of chronic second liver, at least antigen of surface of HBsAg(second liver) ought to be electropositive, and He Xiang's long HBsAg not only it is electronegative, fighting one HBs is electropositive instead, is this why? Original, this is chronic second liver suddenly turn hostile , common ground says, second liver virus does not give a shop sign by convention, hide important second liver virus mark content HBsAg, HBeAg rose. Experts point out, this kind suddenly turn hostile chronic second liver is in clinical on have two kinds of show: One kind is}tBsAg negative, passable examination arrives in blood fight one HBs, perhaps fight one HBe, fight predict HBc; another kind - not only HBsAg is electronegative, all sorts of antibody (be like in front 3 kinds of mentioned antibody) also be electronegative. However, remain essentially the same despite all apparent changes, no matter how change, detect with the most sensitive method HBVDNA, the result is positive still, still put in liver function change, ALT is elevatory. Experts still point out, hide a gender sometimes chronic second liver must pass hepatic puncture, inside hepatic organization ability finds E antigen (HBeAg) , core is fought - former (HBcAg) or HBV DNA. This kind hides - the hair opportunity of survival of chronic second liver makes the gender basically is gene of second liver virus produces mutation (gene of the S before S is mixed mutates) cause, so, be in clinical on, when the doctor lets a patient check HBV DNA to perhaps suggest to do hepatic puncture, the patient should cooperate actively, this is not additional examination project. Suddenly turn hostile 2: HBeAg negative is chronic teacher of king of second liver 　 this year 50 years old, suffer from chronic second liver nearly 20 years, applied content of medium, Western medicine to treat in the past, if using him, say maintain well . He is long disease becomes a good doctor , survive than young doctor to the understanding of chronic second liver much, he is so called all the time small 3 this world (ItBsAg, fight one HBe, fight) of one HBc3 Xiang Weiyang sex, hepatic function is basic and normal, AIT controls wave motion in 40 units. He is quite satisfactory. But feel not quite good recently, apparently lack of power, appetite drops, make water also becomes yellower. Mr. Wang comes to a hospital check, still was before as a result is small 3 this world, however, ALT rises to 350 units however, this Mr. Wang is a little muddleheaded: Small 3 this world are the mark with duplicate blame of second liver virus, since dispute is duplicate, virus of that is to say was restrained, why liver disease made Doctor 　 tell him again: The second liver virus inside your body still is duplicating, because check HBv DNA ration to be 107 copies, milliliter (be less than 103 copies / milliliter duplicates to be not condition) , clew virus is duplicate and active, you liver of this kind of second calls HBeA sex chronic second liver. 　 this is chronic second liver suddenly turn hostile the disaster that cause. This is planted now suddenly turn hostile second liver is very much see, but the attention that did not cause everybody however, some doctors also do not understand very. Announce recently guideline of prevention and cure of chronic hepatitis B it is cent of chronic second liver two kinds: One kind is HBeAg positive chronic second liver, it is everybody those who be familiar with is big liver of 3 in relief second (HBsAg, HBeAg, fight one HBc3 Xiang Weiyang quality) ; is HBeAg negative another kind chronic second liver. Also cry small liver of 3 in relief second, it is the liver of this kind of second that Mr. Wang gets. Liver of second of this one type also must check diagnose HBV DNA. This kind suddenly turn hostile second liver is like next characteristics: 1. Course of diseases is long at HBeA positive chronic second liver, the man with older age sees more. 2. Virus but durative or intermittence is duplicate, so, detect when the HBVDNA in blood, its level often shows fluctuant pattern, on any account, tall when can be more than 108 copies / milliliter, low when can be 104 shellfish / milliliter. When liver of diagnostic HBeAg electronegative second, made clear HBVDNA to be more than or be equal to shellfish of 104 beat / milliliter can be diagnosed, and when liver of second of diagnostic}lBeAg positive, ask HBV I)NA must is more than 10 ' copy / milliliter. 3. ALT of serum of this kind of patient is not very tall. Won't be worth upper limit normally more than commonly 10 times (normal value upper limit is 40 units) , great majority is normal value upper limit 5 times the left and right sides. 4. Illness spontaneity is reduced is scarce very, often have serious liver cell inflammation, necrotic, apparent liver fiber is changed, make an appointment with 409'6 to liver cirrhosis is behaved. Without giving thought to chronic second liver whether suddenly turn hostile , the patient must coach in specialized subject doctor next cure. Choose which kinds of drug, how application, period of treatment how long, how to stop the medicine, note after stopping drug to wait, want to follow doctor's advice. Abuse folk prescription, proved recipe, meeting bungle illness, even lose treats an opportunity. (the exercitation edits: Plum apricot)